Severe tricuspid valve destruction due to untreated endocarditis
Maryem Assamti, Noha Elouafi
Corresponding author: Maryem Assamti, Cardiology Department, Faculty of Medicine and Pharmacy, Mohamed First University, Oujda, Morocco
Received: 22 Aug 2020 - Accepted: 16 Sep 2020 - Published: 21 Sep 2020
Domain: Cardiology
Keywords: Tricuspid endocarditis, valve destruction, heart failure
©Maryem Assamti et al. PAMJ Clinical Medicine (ISSN: 2707-2797). This is an Open Access article distributed under the terms of the Creative Commons Attribution International 4.0 License (https://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
Cite this article: Maryem Assamti et al. Severe tricuspid valve destruction due to untreated endocarditis. PAMJ Clinical Medicine. 2020;4:33. [doi: 10.11604/pamj-cm.2020.4.33.25690]
Available online at: https://www.clinical-medicine.panafrican-med-journal.com//content/article/4/33/full
Severe tricuspid valve destruction due to untreated endocarditis
Maryem Assamti1,&, Noha Elouafi1,2
&Corresponding author
A 23 years old previously healthy man, admitted for tricuspid infective endocarditis complicated with severe tricuspid regurgitation and multiple septic pulmonary embolism (A and B). Blood cultures grew multiresistant (Enterobacter cloacae). He was treated with Imipenem 3g per day with Amikacin 900mg per day for 22 days. Given the uncontrolled infection and the worsening of the tricuspid regurgitation, surgery was indicated, but was not performed due to lack of means. After his discharge, the patient was admitted several times for acute right heart failure. Two years follow up echocardiography showed a destruction of the tricuspid valve, dilatation of the right cavities, and paradoxical motion of the septum (C,D). Tricuspid valve infective endocarditis represents 5 to 10% of all infective endocarditis (IE) cases. In 70-85% of cases, it is successfully treated conservatively, and surgery is only considered for large vegetation with recurrent septic pulmonary emboli, persistent bacteremia, and less often for severe tricuspid regurgitation (TR) and heart failure. Complete tricuspid valve endocarditic destruction is rarely described in the literature, and may lead to irreversible severe right heart failure. This case highlights the necessity of early and prompt medical and surgical management to reduce morbidity related to this condition.
Figure 1: A) four-chamber TTE 2D view showing a vegetation (red arrow) on the tricuspid valve (first admission). RA: right atrium, RV: right ventricle, LV: left ventricle, LA: left atrium; B) four-chamber TTE view showing a severe tricuspid regurgitation on the Color Doppler (first admission); C) four-chamber TTE view showing a severe tricuspid destruction, dilatation of the right cavities and paradoxical motion of the septum (two years follow up). RA: right atrium, RV: right ventricle; D) sub costal view showing dilated inferior vena cave (at 50mm) on the 2 years follow-up TTE. RA: right atrium, IVC: inferior vena cava